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Word Count: 307
Preterm infants are susceptible to disruptions of acid-base digestion with a tendency to metabolic acidosis due to the short-term age-related low kidney capacity for net acid excretion NEA Premature formulas have a high renal NAE thus decreasing their low age-specific reserve quantity of acid excretion This is why preterm infants are at risk of naturally developing a positive acid balance Whereas feeding premature infants human milk results in a low NAE With this being said preterm infants with physiologically impaired renal function are at a risk of developing latent metabolic acidosis A study was then undertaken from 1993 to 1994 in a Dortmund pediatric clinic where 82 premature infants that weighed less than 20 kg were either fed formula or human milk to obtain essential acid-base data in their blood and urine in order to delimit the impact of food mineral composition and kidney regulation on universal acid-base disorders 48 preterm infants were fed with their own mothers milk which was broken into 28 preterm infants were fed natural human milk and 20 were fed with enriched human milk with added fortifiers Since the nutritional needs minerals and proteins of premature infants surpass the content of human milk fortifiers provide those minerals and proteins 34 preterm infants were fed formula with 23 on a standard batch and 11 on an altered batch with reduced acid capacity All premature infants were fed according to the regular schedule of the clinic with an additional supplement of vitamin D Time urine samples were also collected regularly between 6 and 8 am for 8-12 hoursThe data collected during the study showed that the useable energy of fortified human milk was the highest followed by formula with native milk having the lowest values Premature infants had a high renal net acid excretion on standard formula However preterm infants on modified formula with reduced acid load had the lowest renal net acid excretion
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